年輕水球運動員的肌肉損傷
通過在訓練前每天進行的200m最大游泳(P200)和30s橫桿跳躍測試(30CJ)測量性能,並且在最終LLLT干預之前和之後抽取血樣以測量白細胞介素(IL) 和肌肉損傷標記。 與安慰劑組相比,LLLT組的P200運動沒有顯著變化,但30CJ有輕微改善(8.7±2.6%)。 與前,0和24小時相比,IL-1β和腫瘤壞死因子-α在最後一次LLLT干預後48小時內出現增加(P <0.016)濃度,但在各組之間沒有差異。 與LLLT組相比,安慰劑組中IL-10隨時間增加並且達到中等效果。 在LLLT治療組內,肌酸激酶隨時間顯著下降(P = 0.049) Performance was measured by a 200-m maximal swimming (P200) and a 30-s crossbar jump test (30CJ) which was performed every day before training, and blood samples were drawn pre and post the final LLLT intervention to measure interleukins (IL) and muscle damage markers. There was no significant change in the P200 exercise in the LLLT group compared with the placebo group but there was a moderate improvement in the 30CJ (8.7 ± 2.6 %). IL-1β and tumor necrosis factor-alpha presented increased ( P < 0.016) concentration within group 48 h after the last LLLT intervention compared to pre, 0, and 24 h, but did not differ between groups. IL-10 increased over time in the placebo group and reache...